Since the advent of low-cost accelerometers, like the one in near every modern phone, the data obtained from studies of exercise has improved greatly. In the scientific world of the study of exercise and aging, in which it can take a decade or two for enough epidemiological evidence to accumulate to change minds, accelerometers are still a comparatively recent innovation. The study noted here is an example of the sort of work being accomplished in this context. Like most such studies, the data strongly suggests that exercise slows the onset of cardiovascular aging, and thus lowers the risk of cardiovascular disease.
Should we view it as a failure of the established, mainstream approach to research and development of therapies to treat age-related disease that exercise remains one of the best and most reliable options on the table? Quite possibly. This is an era of accelerating, revolutionary progress in the tools and capabilities of biotechnology. The research community should have achieved far more than it has to date. The failure to do so is arguably due to the adoption of an ineffective strategy, one that largely revolves around attempts to adjust the late stage disease state rather than seeking to address
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